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The proposed mechanism involves TB-500s regulation of the actin cytoskeleton during the remodeling phase of wound healing, where the balance between myofibroblast contraction (which drives scar formation) and normal cell migration and differentiation (which drives regeneration) determines tissue outcome
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It acts on fibroblasts to stimulate collagen and elastin production, accelerates wound healing and tissue remodeling, supports hair follicle cycling, and has well-documented anti-inflammatory effects across multiple tissue types
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