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Mechanistically, pterostilbene treatment of RBL-2H3 cells increased phosphorylation of Serine/Threonine Kinase 11 (LKB1), AMPK, and acetyl co-A carboxylase, and decreased proteins involved in FcRI-mediated signaling, including phosphorylated p38, ERK, JNK, IKK and mTOR, suggesting pterostilbene both activates the LKB1/AMPK pathway and inhibits FcRI signaling [209]

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